mouse anti phospho p44 42 mapk perk (Cell Signaling Technology Inc)
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mouse anti phospho p44 42 mapk perk
Mouse Anti Phospho P44 42 Mapk Perk, supplied by Cell Signaling Technology Inc, used in various techniques. Bioz Stars score: 86/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/mouse+anti-perk/pmc13007337-12-0-5
Average 86 stars, based on 1 article reviews
Mouse Anti Phospho P44 42 Mapk Perk, supplied by Cell Signaling Technology Inc, used in various techniques. Bioz Stars score: 86/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/mouse+anti-perk/pmc13007337-12-0-5
Average 86 stars, based on 1 article reviews
mouse anti phospho p44 42 mapk perk - by Bioz Stars,
2026-09
86/100 stars
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Purification:Article Title: REQUIREMENT OF 3-PHOSPHOINOSITIDE-DEPENDENT PROTEIN KINASE-1 (PDK1) FOR BDNF-MEDIATED NEURONAL SURVIVAL Article Snippet: For pERK and pRSK immunostaining, Article Title: ERK is involved in tooth-pressure-induced Fos expression in Vc neurons. Article Snippet: Discomfort and pain encountered during orthodontic treatment are major problems for patients, but the details of the underlying neural processes and molecular mechanisms are not well-understood.. Here we show that noxious tooth mechanical pressure induced by orthodontic elastics resulted in a rapid and transient activation of extracellular signal-regulated protein kinase (ERK) in the trigeminal spinal subnucleus interpolaris and caudalis transition zone (Vi/Vc), trigeminal spinal subnucleus caudalis (Vc), and upper cervical spinal cord (Vc/C2).. The phosphorylated ERK (pERK) was observed in neurons but not in astroglia and microglia. Article Title: MarvelD3 couples tight junctions to the MEKK1–JNK pathway to regulate cell behavior and survival Article Snippet: Antibodies against other proteins were as previously described or were obtained from commercial sources: rabbit anti-MEKK1 (Abcam); mouse anti-pJNK, rabbit anti-JNK, mouse anti-pErk, rabbit anti-Erk, and rabbit anti-pp38 and - Article Title: Therapeutic effects of human mesenchymal stem cells on traumatic brain injury in rats: secretion of neurotrophic factors and inhibition of apoptosis. Article Snippet: To investigate the therapeutic effects and mechanisms of action of human mesenchymal stem cells (hMSCs), rats were intravenously treated with hMSCs 24 h after traumatic brain injury (TBI).. Neurological function was significantly recovered in the hMSC-treated group by 15 days after TBI compared to the placebo group treated with saline.. Quantitative ELISA of extracts from the entire traumatized cerebral hemispheres showed significantly increased expression of nerve growth factor (NGF), brain-derived neurotrophic factor (BDNF), and neurotrophin-3 (NT-3) in the hMSC group at 2 days after TBI, with expression decreasing over time. Recombinant:Article Title: REQUIREMENT OF 3-PHOSPHOINOSITIDE-DEPENDENT PROTEIN KINASE-1 (PDK1) FOR BDNF-MEDIATED NEURONAL SURVIVAL Article Snippet: For pERK and pRSK immunostaining, Article Title: ERK is involved in tooth-pressure-induced Fos expression in Vc neurons. Article Snippet: Discomfort and pain encountered during orthodontic treatment are major problems for patients, but the details of the underlying neural processes and molecular mechanisms are not well-understood.. Here we show that noxious tooth mechanical pressure induced by orthodontic elastics resulted in a rapid and transient activation of extracellular signal-regulated protein kinase (ERK) in the trigeminal spinal subnucleus interpolaris and caudalis transition zone (Vi/Vc), trigeminal spinal subnucleus caudalis (Vc), and upper cervical spinal cord (Vc/C2).. The phosphorylated ERK (pERK) was observed in neurons but not in astroglia and microglia. Article Title: MarvelD3 couples tight junctions to the MEKK1–JNK pathway to regulate cell behavior and survival Article Snippet: Antibodies against other proteins were as previously described or were obtained from commercial sources: rabbit anti-MEKK1 (Abcam); mouse anti-pJNK, rabbit anti-JNK, mouse anti-pErk, rabbit anti-Erk, and rabbit anti-pp38 and - Article Title: Therapeutic effects of human mesenchymal stem cells on traumatic brain injury in rats: secretion of neurotrophic factors and inhibition of apoptosis. Article Snippet: To investigate the therapeutic effects and mechanisms of action of human mesenchymal stem cells (hMSCs), rats were intravenously treated with hMSCs 24 h after traumatic brain injury (TBI).. Neurological function was significantly recovered in the hMSC-treated group by 15 days after TBI compared to the placebo group treated with saline.. Quantitative ELISA of extracts from the entire traumatized cerebral hemispheres showed significantly increased expression of nerve growth factor (NGF), brain-derived neurotrophic factor (BDNF), and neurotrophin-3 (NT-3) in the hMSC group at 2 days after TBI, with expression decreasing over time. Isolation:Article Title: REQUIREMENT OF 3-PHOSPHOINOSITIDE-DEPENDENT PROTEIN KINASE-1 (PDK1) FOR BDNF-MEDIATED NEURONAL SURVIVAL Article Snippet: For pERK and pRSK immunostaining, Article Title: ERK is involved in tooth-pressure-induced Fos expression in Vc neurons. Article Snippet: Discomfort and pain encountered during orthodontic treatment are major problems for patients, but the details of the underlying neural processes and molecular mechanisms are not well-understood.. Here we show that noxious tooth mechanical pressure induced by orthodontic elastics resulted in a rapid and transient activation of extracellular signal-regulated protein kinase (ERK) in the trigeminal spinal subnucleus interpolaris and caudalis transition zone (Vi/Vc), trigeminal spinal subnucleus caudalis (Vc), and upper cervical spinal cord (Vc/C2).. The phosphorylated ERK (pERK) was observed in neurons but not in astroglia and microglia. Article Title: MarvelD3 couples tight junctions to the MEKK1–JNK pathway to regulate cell behavior and survival Article Snippet: Antibodies against other proteins were as previously described or were obtained from commercial sources: rabbit anti-MEKK1 (Abcam); mouse anti-pJNK, rabbit anti-JNK, mouse anti-pErk, rabbit anti-Erk, and rabbit anti-pp38 and - Article Title: Therapeutic effects of human mesenchymal stem cells on traumatic brain injury in rats: secretion of neurotrophic factors and inhibition of apoptosis. Article Snippet: To investigate the therapeutic effects and mechanisms of action of human mesenchymal stem cells (hMSCs), rats were intravenously treated with hMSCs 24 h after traumatic brain injury (TBI).. Neurological function was significantly recovered in the hMSC-treated group by 15 days after TBI compared to the placebo group treated with saline.. Quantitative ELISA of extracts from the entire traumatized cerebral hemispheres showed significantly increased expression of nerve growth factor (NGF), brain-derived neurotrophic factor (BDNF), and neurotrophin-3 (NT-3) in the hMSC group at 2 days after TBI, with expression decreasing over time. Expressing:Article Title: REQUIREMENT OF 3-PHOSPHOINOSITIDE-DEPENDENT PROTEIN KINASE-1 (PDK1) FOR BDNF-MEDIATED NEURONAL SURVIVAL Article Snippet: For pERK and pRSK immunostaining, Article Title: ERK is involved in tooth-pressure-induced Fos expression in Vc neurons. Article Snippet: Discomfort and pain encountered during orthodontic treatment are major problems for patients, but the details of the underlying neural processes and molecular mechanisms are not well-understood.. Here we show that noxious tooth mechanical pressure induced by orthodontic elastics resulted in a rapid and transient activation of extracellular signal-regulated protein kinase (ERK) in the trigeminal spinal subnucleus interpolaris and caudalis transition zone (Vi/Vc), trigeminal spinal subnucleus caudalis (Vc), and upper cervical spinal cord (Vc/C2).. The phosphorylated ERK (pERK) was observed in neurons but not in astroglia and microglia. Article Title: MarvelD3 couples tight junctions to the MEKK1–JNK pathway to regulate cell behavior and survival Article Snippet: Antibodies against other proteins were as previously described or were obtained from commercial sources: rabbit anti-MEKK1 (Abcam); mouse anti-pJNK, rabbit anti-JNK, mouse anti-pErk, rabbit anti-Erk, and rabbit anti-pp38 and - Article Title: Therapeutic effects of human mesenchymal stem cells on traumatic brain injury in rats: secretion of neurotrophic factors and inhibition of apoptosis. Article Snippet: To investigate the therapeutic effects and mechanisms of action of human mesenchymal stem cells (hMSCs), rats were intravenously treated with hMSCs 24 h after traumatic brain injury (TBI).. Neurological function was significantly recovered in the hMSC-treated group by 15 days after TBI compared to the placebo group treated with saline.. Quantitative ELISA of extracts from the entire traumatized cerebral hemispheres showed significantly increased expression of nerve growth factor (NGF), brain-derived neurotrophic factor (BDNF), and neurotrophin-3 (NT-3) in the hMSC group at 2 days after TBI, with expression decreasing over time. Bioprocessing:Article Title: REQUIREMENT OF 3-PHOSPHOINOSITIDE-DEPENDENT PROTEIN KINASE-1 (PDK1) FOR BDNF-MEDIATED NEURONAL SURVIVAL Article Snippet: For pERK and pRSK immunostaining, Article Title: ERK is involved in tooth-pressure-induced Fos expression in Vc neurons. Article Snippet: Discomfort and pain encountered during orthodontic treatment are major problems for patients, but the details of the underlying neural processes and molecular mechanisms are not well-understood.. Here we show that noxious tooth mechanical pressure induced by orthodontic elastics resulted in a rapid and transient activation of extracellular signal-regulated protein kinase (ERK) in the trigeminal spinal subnucleus interpolaris and caudalis transition zone (Vi/Vc), trigeminal spinal subnucleus caudalis (Vc), and upper cervical spinal cord (Vc/C2).. The phosphorylated ERK (pERK) was observed in neurons but not in astroglia and microglia. Article Title: MarvelD3 couples tight junctions to the MEKK1–JNK pathway to regulate cell behavior and survival Article Snippet: Antibodies against other proteins were as previously described or were obtained from commercial sources: rabbit anti-MEKK1 (Abcam); mouse anti-pJNK, rabbit anti-JNK, mouse anti-pErk, rabbit anti-Erk, and rabbit anti-pp38 and - Article Title: Therapeutic effects of human mesenchymal stem cells on traumatic brain injury in rats: secretion of neurotrophic factors and inhibition of apoptosis. Article Snippet: To investigate the therapeutic effects and mechanisms of action of human mesenchymal stem cells (hMSCs), rats were intravenously treated with hMSCs 24 h after traumatic brain injury (TBI).. Neurological function was significantly recovered in the hMSC-treated group by 15 days after TBI compared to the placebo group treated with saline.. Quantitative ELISA of extracts from the entire traumatized cerebral hemispheres showed significantly increased expression of nerve growth factor (NGF), brain-derived neurotrophic factor (BDNF), and neurotrophin-3 (NT-3) in the hMSC group at 2 days after TBI, with expression decreasing over time. Control:Article Title: REQUIREMENT OF 3-PHOSPHOINOSITIDE-DEPENDENT PROTEIN KINASE-1 (PDK1) FOR BDNF-MEDIATED NEURONAL SURVIVAL Article Snippet: For pERK and pRSK immunostaining, Article Title: ERK is involved in tooth-pressure-induced Fos expression in Vc neurons. Article Snippet: Discomfort and pain encountered during orthodontic treatment are major problems for patients, but the details of the underlying neural processes and molecular mechanisms are not well-understood.. Here we show that noxious tooth mechanical pressure induced by orthodontic elastics resulted in a rapid and transient activation of extracellular signal-regulated protein kinase (ERK) in the trigeminal spinal subnucleus interpolaris and caudalis transition zone (Vi/Vc), trigeminal spinal subnucleus caudalis (Vc), and upper cervical spinal cord (Vc/C2).. The phosphorylated ERK (pERK) was observed in neurons but not in astroglia and microglia. Article Title: MarvelD3 couples tight junctions to the MEKK1–JNK pathway to regulate cell behavior and survival Article Snippet: Antibodies against other proteins were as previously described or were obtained from commercial sources: rabbit anti-MEKK1 (Abcam); mouse anti-pJNK, rabbit anti-JNK, mouse anti-pErk, rabbit anti-Erk, and rabbit anti-pp38 and - Article Title: Therapeutic effects of human mesenchymal stem cells on traumatic brain injury in rats: secretion of neurotrophic factors and inhibition of apoptosis. Article Snippet: To investigate the therapeutic effects and mechanisms of action of human mesenchymal stem cells (hMSCs), rats were intravenously treated with hMSCs 24 h after traumatic brain injury (TBI).. Neurological function was significantly recovered in the hMSC-treated group by 15 days after TBI compared to the placebo group treated with saline.. Quantitative ELISA of extracts from the entire traumatized cerebral hemispheres showed significantly increased expression of nerve growth factor (NGF), brain-derived neurotrophic factor (BDNF), and neurotrophin-3 (NT-3) in the hMSC group at 2 days after TBI, with expression decreasing over time. |
